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His unaffected mother was heterozygous for the mutation, which was not present in an unaffected sib; no DNA from the father was available.
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Similar to the p85 IP, the SHP2 IP showed that the p85-SHP2 interaction was abrogated when BCR-ABL pathway was inhibited with imatinib, a result suggesting that ERK may be connected to the p85-SHP2 complex.
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